1Department of NMR, All India Institure of Medical Sciences, New Delhi, India
2Department of NMR, All India Institute of Medical Sciences, New Delhi, India
3Department of Neurology, All India Institute of Medical Sciences, New Delhi, India
4All India Institute of Medical Sciences, New Delhi, India
Presenting Author: Arpita Jaiswal
Synopsis
Motivation:
Goals:
Approach:
Results:
Full abstract & presentation
The full text, figures, and any recorded presentation for this abstract are not shown here. Log in if you are a member or registered attendee with access.
Full abstracts, figures, and presentations for Cape Town - 2026 ISMRM-ISMRT Annual Meeting and Exhibition are available to registered attendees. This content becomes freely available to the public roughly two years after the meeting.
To request or purchase access, contact the ISMRM Central Office at info@ismrm.org.
1. O'Keeffe GW, Sullivan AM. Evidence for dopaminergic axonal degeneration as an early pathological process in Parkinson's disease. Parkinsonism Relat Disord. 2018;56:9-15. doi:10.1016/j.parkreldis.2018.06.025 [doi]
2. Magrinelli F, Picelli A, Tocco P, et al. Pathophysiology of Motor Dysfunction in Parkinson's Disease as the Rationale for Drug Treatment and Rehabilitation. Parkinsons Dis. 2016;2016:9832839. doi:10.1155/2016/983283 [doi]
3. Castelli S, Carinci E, Baldelli S. Oxidative Stress in Neurodegenerative Disorders: A Key Driver in Impairing Skeletal Muscle Health. Int J Mol Sci. 2025;26(12):5782. doi:10.3390/ijms26125782 [doi]
4. Gu W, He C, Chen J, Li J. Proton Magnetic Resonance Spectroscopy for the Early Diagnosis of Parkinson's Disease in the Substantia Nigra and Globus Pallidus: A Meta-Analysis With Trial Sequential Analysis. Front Neurol. 2022;13:838230. doi:10.3389/fneur.2022.838230 [doi]
5. Petiet A. Current and Emerging MR Methods and Outcome in Rodent Models of Parkinson's Disease: A Review. Front Neurosci. 2021;15:583678. doi:10.3389/fnins.2021.583678 [doi]
6. Im HJ, Hahm J, Kang H, et al. Disrupted brain metabolic connectivity in a 6-OHDA-induced mouse model of Parkinson's disease examined using persistent homology-based analysis. Sci Rep. 2016;6:33875. doi:10.1038/srep33875 [doi]
7. Qiu J, Peng G, Tang Y, et al. Lipid profiles in the cerebrospinal fluid of rats with 6-hydroxydopamine-induced lesions as a model of Parkinson's disease. Front Aging Neurosci. 2023;14:1077738. doi:10.3389/fnagi.2022.1077738 [doi]
8. Jang DP, Min HK, Lee SY, et al. Functional neuroimaging of the 6-OHDA lesion rat model of Parkinson's disease. Neurosci Lett. 2012;513(2):187-192. doi:10.1016/j.neulet.2012.02.034 [doi]
9. Tanguay W, Ducrot C, Giguère N, Bourque MJ, Trudeau LE. Neonatal 6-OHDA lesion of the SNc induces striatal compensatory sprouting from surviving SNc dopaminergic neurons without VTA contribution. Eur J Neurosci. 2021;54(7):6618-6632. doi:10.1111/ejn.15437 [doi]
10. Tancheva LP, Lazarova MI, Alexandrova AV, et al. Neuroprotective Mechanisms of Three Natural Antioxidants on a Rat Model of Parkinson's Disease: A Comparative Study. Antioxidants (Basel). 2020;9(1):49. doi:10.3390/antiox9010049 [doi]
11. Carvalho MM, Campos FL, Coimbra B, et al. Behavioral characterization of the 6-hydroxidopamine model of Parkinson's disease and pharmacological rescuing of non-motor deficits. Mol Neurodegener. 2013;8:14. doi:10.1186/1750-1326-8-14 [doi]